Parenchymal senyongo: iimpawu, izinto, isifo

Namhlanje kukho izifo ezininzi ezahlukeneyo ukuba abantu ayikho rhoqo kakhulu. Kodwa kufuneka uyazi kubo ubuncinane kancinane njalo. Ngoko ke, umzekelo, unako ukuthintela kukuvela iingxaki ezinjalo. Kungenxa yoko le nto namhlanje ndifuna ukukuxelela ngokweenkcukacha kakhulu ngale ngezifo njengoko parenchymal senyongo.

lwesigama

Phambi kokuba bajongane sifo, kuyimfuneko ukuchaza abo magama, ezithi zisetyenziswe kakhulu kweli nqaku. senyongo Ngoko parenchymal - sisifo, kodwa kunoko, uphawu oluthile, nto leyo luphawulwa luyawudyobha ngombala senyongo ayikho eluswini kuphela, kodwa inwebu yangaphakathi (eye sclera). bonke Kuyenzeka kuba isigulane iqala ukuba nomzi ngokuthe yibilirubin (oku inyongo kombala). Kubalulekile ukuqaphela ukuba le uphawu ibonisa ukuba isigulana kuneengxaki isibindi, ngubani ngokukhawuleza kangangoko kunokwenzeka kufuneka ukuyisombulula.

Okubangel 'umdla kukuba amayeza kukho inkolelo yobuxoki senyongo. Kulo mzekelo Akukho yibilirubin oluhlala emzimbeni womntu, kunye carotene, into yendalo leyo kwenzeka ngenxa amabala esikhumbeni. Ngoko ke, inokwenzeka kwimeko rhoqo kakhulu kwaye kukuninzi ukutya iminqathe, amathanga, orenji, kunye beetroot. Le ngxaki kunokwenzeka ukuba ixesha elide ukutya picric acid, kwakunye namanye amayeza iziyobisi.

picture clinical

Njani na lo ngezifo njengoko parenchymal senyongo? Le Pathogenesis kwesifo elandelayo. Ngenxa yoko, ekuqaleni kufuneka kuqatshelwe ukuba ngexesha lomsebenzi eliqhelekileyo, yibilirubin, inyongo sakhi, salawo ngokwemvelo. Ukuba kukho ezinye iingxaki esibindini, ethe ngcembe uqalisa yande emzimbeni, ezimpondweni ibonakaliswa yellowing ulusu kunye ngenwebu yangaphakathi. Ukujonga ulwazi oko kukuthi parenchymal senyongo, kubalulekile kuthi ukuba lo mba awunanto izigaba ezithathu eziphambili zophuhliso.

1. Isigaba sokuqala. Kulo mzekelo, ukuncitshiswa kumsebenzi enqanda, inwebu ezonakeleyo hepatocytes, yibilirubin ngqo oyekayo ngokuqhelekileyo zaveliswa.
2. Xa Inqanaba lesibini sifo izigulane unengxaki efana giperfermentiya, hepatocyte inwebu permeability ukunyuka. Intlungu ecaleni layo ivela njenge ngenxa ucinezelo le emithanjeni.
3. Xa inyathelo wesithathu nkqubo a glucuronidation ukuphazamiseka yibilirubin lwenzeka ukusilela hepatic. Kwakhona, umchamo uba ngakhanyi kakhulu, iintlungu rhoqo waziva ecaleni kwakhe.

Kufuneka kuqatshelwe ukuba kubalulekile ukuqalisa ukujongana parenchymal senyongo kumanqanaba zokuqala ingenzeka sifo. Eneneni, kulo mzekelo, zijongene le ngxaki kuba lula yaye ngokukhawuleza kakhulu. Ukuba i "ukulibala" sisiqalo senkqubo yokuphilisa ukuya kwisigaba sesithathu, le unyango kuya kuba nzima kakhulu ukuba esi sifo.

Umthombo le ngxaki

Kutheni angavela parenchymal senyongo, zithini izizathu eziphambili ukuba ivela khona?

• Okokuqala kufanele kuqatshelwe ukuba ngxaki iphambili kulo mzekelo ezi arhente ezisulelayo uthi: iintsholongwane, iintsholongwane, abulale. Amaninzi, kulo meko sithetha izifo ezifana seengcongconi, negcushuwa, toxoplasmosis kunye nabanye.
• Hlula gepatotsidov tyhefu zombini eziphilayo nezingaphiliyo. ityhefu yityhefu umzimba zingenzeka ngenxa ukusebenzisa of ukudliwa, utywala libya-umgangatho, emva kokufumana iarseniki okanye inyoka ukuluma.
• Unobangela ingxaki nako ukuthatha izinto ezithile amayeza kulo neziphumo hepatotoxic. Kulo mzekelo, amaxesha amaninzi kufikwa ethile efana "paracetamol" okanye antibiotics.

ufuzo

Ukuqwalasela ezibangela parenchymal senyongo, kufuneka kwakhona kwakhona bathi esi sifo nalo into njengelifa. Oko kukuthi, oko sigqithiswe uhlobo ukusuka ilungu losapho omnye komnye.

• Mhlawumbi ukusilela ligandinov-ezithile ziiproteni ezibekwe kumphezulu isibindi.
• Isizathu abe ukuswela ilifa enzayim esithile, ekuthiwa UDP-glucuronyl.
• Ngoko ke ilifa, kunye nesifo Wilson kaThixo, xa kukho zemali kwi hepatocytes zobhedu, ngenxa yale siphene yemfuza iphazamisekile ukusebenza komzimba.

The main Iimpawu

Ukuqwalasela izifo ezifana iimpawu parenchymal senyongo - yiloo nto futhi udinga ukuxelelwa. Ngoko ke zintoni ukubonakalalisa na esi sifo?

1. Ubungqina yokuqala yellowing ingxaki ulusu kunye uxakaxa imifinya, ikakhulu amehlo sclera.
2. Kwakhona, isigulane inokuba ukurhawuzelela wobulali.
3. Maxa wambi intloko ebuhlungu, ifiva, intloko ebuhlungu.
4. Kaninzi izigulane akhalaze zokulahleka wokutya, neziphumo umzimba kwenzeka.
5. Kwakhona, kukho intlungu kwezihlunu kunye ubuthathaka jikelele.

Izalathisi ihlaziya nazo azifani.

• Endleni uba eqaqambileyo, yabangazijongisisi kancinane. Yonke ilahleko kwenzeka stercobilin ngenxa, omnye ngombala cholic.
• Umchamo kwakhona itshintsha umbala. Kungaba zombini ukukhanya kakhulu kwaye kumnyama kakhulu.
• Egazini izigulane kwandisa umxholo yibilirubin ngqo nengxelo-ntetho.

ekuxilongweni

Njengoko kuqondwe ukuba umntu parenchymal senyongo? Uxilongo - ukuze Bothetha ubukho besi sifo. Ukuze wenze oku:

1. Ukwenza uhlalutyo jikelele umchamo, endleni kunye negazi.
2. Uvavanyo womfanekiso lwezonyango jikelele.
3. Uhlalutyo ezinto igazi.
4. ezinye iimvavanyo instrumental linokukwenziwa.

iimvavanyo clinical

Ukuba isigulana eso sathathwa analysis of endleni kunye nomchamo, kulo mzekelo, xa izikhombisi yibilirubin izifo urobilin linyuke kakhulu, ngelixa inani stercobilin uyawa, nto leyo ebangela imibala buthathaka ilindle.

Kwakhona, iimvavanyo zegazi iza kufuneka. Kulo mzekelo, kusenokubakho leukocytosis kunye nezinye. Ukuba parenchymal senyongo nomonde, ngamachiza igazi - oko kubalulekile. Apha yokusebenza ebotshiwe yibilirubin engadityaniswanga kunye urobilin baya kuvuswa.

Kwezinye iimeko, isigulane liya kuqesha nezinye zophando ezongeziweyo. Oku ingaba:

• Ukubekwa folic acid kunye vitamin B12 amanqanaba egazini.
• Ukumiselwa amakishayo ithumba.
• Coprogram: Uhlalutyo-gazi kunye iintshulube.

izifundo instrumental

Lomntu ke onobu bugcisa abanezakhono unakho, ngaphandle naziphi na iingxaki ezithile ukwahlula parenchymal senyongo syndrome kwizifo ngokupheleleyo. Noko ke, kwezinye iimeko, nkqu izikhanyiso baziincamisa zezonyango ukuqinisekisa ukuziphatha kwesifo izifundo ezongezelelweyo ezifunekayo isixhobo. Kulo mzekelo, isigulana unako ufakwe:

1. Ray. Ukuqaphela iingxaki kunye esiswini, kuvuleke iminxunya namathumbu.
2. Cholecystography. Kuyimfuneko ukuba siqonde ukuba kukho amatye inyongo isinye.
3. Ultrasound uyakwazi ukunika inkcazelo malunga nemeko wesibindi kunye nezinye izigqeba ezinkulu.
4. CT okanye MRI.

kunqabile kakhulu, izigulane ezilawulwa nokuba biopsy wesibindi.

unyango

Kufuneka kuqatshelwe kanjalo ukuba esi ngezifo na iindlela ezininzi yokulahla. Kuxhomekeke iqondo ukungahoywa sifo ngxabano. Amaninzi, oogqirha amisele iziyobisi ukususa yibilirubin agqithisileyo emzimbeni. Noko ke, asingabo bonke iyeza kunceda. Kulo mzekelo, ugqirha ukuze amisele inkqubo ezifana phototherapy. Kwaye sonke ngoba ukukhanya yelanga unako ukwahlukana ngokukhawuleza ezo molekyuli yibilirubin nokunceda ukususwa yazo ngokukhawuleza. Kule sifo kukwabalulekile ukuba sithathe iivithamini ezintsonkothileyo, ingakumbi B no-C, lipoic acid kunye acid.

Prevention and Iingxaki

Ekubeni parenchymal senyongo - luphawu owenzeke imvelaphi nezinye izifo (viral hepatitis B no-C, amatye isinyi, njalo-njalo), ngoku lixesha kubaluleke kakhulu ukuqala ukuphelisa ezi zifo. Ngapha koko, simele sikhumbule ukuba ixesha ayiboneleli unonophelo nomonde, oku kunokuphumela iingxaki ezithile. Oko kukuthi, isiguli ngokusisigxina ziphazamise isibindi nenyongo lwesinye. Kulo mzekelo, ukufumaneka ukwanda kwe yibilirubin ebangela ukunxila, nto leyo nefuthe elibi kakhulu kumsebenzi onke amalungu kunye neenkqubo zayo.